Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/106709
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dc.contributor.authorGagliardi, L.-
dc.contributor.authorBurt, M.-
dc.contributor.authorFeng, J.-
dc.contributor.authorPoplawski, N.-
dc.contributor.authorScott, H.-
dc.date.issued2016-
dc.identifier.citationClinical Endocrinology, 2016; 85(3):495-497-
dc.identifier.issn0300-0664-
dc.identifier.issn1365-2265-
dc.identifier.urihttp://hdl.handle.net/2440/106709-
dc.descriptionLetter to the Editor-
dc.description.statementofresponsibilityLucia Gagliardi, Morton G. Burt, Jinghua Feng, Nicola K. Poplawski and Hamish S. Scott-
dc.language.isoen-
dc.publisherWiley-Blackwell-
dc.rights© 2016 John Wiley & Sons Ltd-
dc.source.urihttp://dx.doi.org/10.1111/cen.13104-
dc.subjectHumans-
dc.subjectHypocalcemia-
dc.subjectReceptors, Calcium-Sensing-
dc.subjectPedigree-
dc.subjectMutation-
dc.subjectMiddle Aged-
dc.subjectFemale-
dc.subjectFractures, Bone-
dc.titleAutosomal dominant hypocalcaemia due to a novel CASR mutation: clinical and genetic implications-
dc.typeJournal article-
dc.identifier.doi10.1111/cen.13104-
dc.relation.granthttp://purl.org/au-research/grants/nhmrc/1023059-
pubs.publication-statusPublished-
dc.identifier.orcidPoplawski, N. [0000-0002-9372-3325]-
dc.identifier.orcidScott, H. [0000-0002-5813-631X]-
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