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Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/19205
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dc.contributor.authorJenkins, Brendan Johnen
dc.date.issued1998en
dc.identifier.urihttp://hdl.handle.net/2440/19205-
dc.descriptionIncludes bibliographical references (17 leaves)en
dc.descriptionvii, 113, [89] leaves, [32] leaves of plates : ill. (some col.) ; 30 cm.en
dc.description.abstractGenerates and exploits activating point mutations in the common β subunit of the human GM-CSF, IL-3 and IL-5 receptors. Elucidates the mechanisms by which the isolated mutations confer constitutive activity on hβc. Identifies the putative all-specific signalling molecules and provides a map of the locations of activating point mutations in hβc,en
dc.format.extent111608 bytesen
dc.format.mimetypeapplication/pdfen
dc.language.isoenen
dc.subject.lcshOncogenes.en
dc.subject.lcshHematopoiesis.en
dc.subject.lcshCytokines Receptors.en
dc.titleActivating point mutations in the common β subunit of the human GM-CSF, IL-3 and IL-5 receptors : implications for receptor function and role in disease / by Brendan John Jenkins.en
dc.typeThesisen
dc.contributor.schoolDept. of Medicineen
dc.provenanceThis electronic version is made publicly available by the University of Adelaide in accordance with its open access policy for student theses. Copyright in this thesis remains with the author. This thesis may incorporate third party material which has been used by the author pursuant to Fair Dealing exception. If you are the author of this thesis and do not wish it to be made publicly available or If you are the owner of any included third party copyright material you wish to be removed from this electronic version, please complete the take down form located at: http://www.adelaide.edu.au/legalsen
dc.description.dissertationThesis (Ph.D.)--University of Adelaide, Dept. of Medicine, 1998en
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