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|Title:||Cross-resistance to imazethapyr in a fluazifop-P-butyl-resistant population of Digitaria sanguinalis|
|Citation:||Pesticide Biochemistry and Physiology, 2001; 71(3):190-195|
|Publisher:||Academic Press Inc|
|Imam Hidayat and Christopher Preston|
|Abstract:||A population of Digitaria sanguinalis selected by and resistant to the acetyl-coenzyme A carboxylase-inhibiting herbicide fluazifop-P-butyl is cross-resistant to the acetolactate synthase (ALS) inhibitor imazethapyr. The resistant population was six-fold resistant to imazethapyr despite never having been exposed to any ALS-inhibiting herbicide. ALS activity extracted from the resistant population was susceptible to imidazolinone herbicides. Malathion, a known cytochrome P450 monooxygenase inhibitor, synergized imazethapyr activity on the resistant population, suggesting that resistance is due to increased herbicide metabolism. In contrast, malathion antagonized fluazifop-P-butyl activity. Therefore, it is likely that the enzyme responsible for fluazifop acid detoxification in D. sanguinalis is different from the enzyme that detoxifies imazethapyr.|
|Description:||Copyright © 2001 Elsevier Science (USA). All rights reserved.|
|Appears in Collections:||Agriculture, Food and Wine publications|
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