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PreviewIssue DateTitleAuthor(s)
2017Mitochondrion to endoplasmic reticulum apposition length in zebrafish embryo spinal progenitors is unchanged in response to perturbations associated with Alzheimer’s diseaseNewman, M.; Halter, L.; Lim, A.; Lardelli, M.; Lakshmana, M.
2016The enemy within: innate surveillance-mediated cell death, the common mechanism of neurodegenerative diseaseRichards, R.; Robertson, S.; O'Keefe, L.; Fornarino, D.; Scott, A.; Lardelli, M.; Baune, B.
2012Robust homeostasis of Presenilin1 protein levels by transcript regulationNewman, M.; Nornes, S.; Martins, R.; Lardelli, M.
2011Zebrafish as a tool in Alzheimer's disease researchNewman, M.; Verdile, G.; Martins, R.; Lardelli, M.
2011The response of HMGA1 to changes in oxygen availability is evolutionarily conservedMoussavi Nik, S.; Newman, M.; Lardelli, M.
2001Evolutionary analysis of vertebrate Notch genesKortschak, R.; Tamme, R.; Lardelli, M.
2001Tyrosinase gene expression in zebrafish embryosCamp-Dotlic, E.; Lardelli, M.
2014Using the zebrafish model for Alzheimer's disease researchNewman, M.; Ebrahimie, E.; Lardelli, M.
2014Differential, dominant activation and inhibition of Notch signalling and APP cleavage by truncations of PSEN1 in human diseaseNewman, M.; Wilson, L.; Verdile, G.; Lim, A.; Khan, I.; Nik, S.; Pursglove, S.; Chapman, G.; Martins, R.; Lardelli, M.
2007Zebrafish Angiotensin II receptor-like 1a (agtrl1a) is expressed in migrating hypoblast, vasculature, and in multiple embryonic epitheliaTucker, B.; Hepperle, C.; Kortschak, R.; Rainbird, B.; Wells, S.; Oates, A.; Lardelli, M.