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Results 31-40 of 125 (Search time: 0.003 seconds).
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PreviewIssue DateTitleAuthor(s)
1998Molecular analysis of shiga toxigenic Escherichia coli O111:H proteins which react with sera from patients with hemolytic-uremic syndrome.Voss, E.; Paton, A.; Manning, P.; Paton, J.
2009Acquisition of Anergy to Proinflammatory Cytokines in Nonimmune Cells through Endoplasmic Reticulum Stress Response: A Mechanism for Subsidence of InflammationHayakawa, K.; Hiramatsu, N.; Okamura, M.; Yamazaki, H.; Nakajima, S.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.
2000A new biological agent for treatment of Shiga toxigenic Escherichia coli infections and dysentery in humansPaton, A.; Morona, R.; Paton, J.
2009Suppression of NF-kappa B by Cyclosporin A and Tacrolimus (FK506) via Induction of the C/EBP Family: Implication for Unfolded Protein ResponseDu, S.; Hiramatsu, N.; Hayakawa, K.; Kasai, A.; Okamura, M.; Huang, T.; Yao, J.; Takeda, M.; Araki, I.; Sawada, N.; Paton, A.; Paton, J.; Kitamura, M.
2018Long interleukin-22 binding protein isoform-1 is an intracellular activator of the unfolded protein responseGómez-Fernández, P.; Urtasun, A.; Paton, A.; Paton, J.; Borrego, F.; Dersh, D.; Argon, Y.; Alloza, I.; Vandenbroeck, K.
2017Structure aided design of a Neu5Gc specific lectinDay, C.; Paton, A.; Higgins, M.; Shewell, L.; Jen, F.; Schulz, B.; Herdman, B.; Paton, J.; Jennings, M.
2009A subcytotoxic dose of subtilase cytotoxin prevents lipopolysaccharide-induced inflammatory responses, depending on its capacity to induce the unfolded protein responseHarama, D.; Koyama, K.; Mukai, M.; Shimokawa, N.; Miyata, M.; Nakamura, Y.; Ohnuma, Y.; Ogawa, H.; Matsuoka, S.; Paton, A.; Paton, J.; Kitamura, M.; Nakao, A.
2014The cholesterol-dependent cytolysins pneumolysin and streptolysin O require binding to red blood cell glycans for hemolytic activityShewell, L.; Harvey, R.; Higgins, M.; Day, C.; Hartley-Tassell, L.; Chen, A.; Gillen, C.; James, D.; Alonzo, F.; Torres, V.; Walker, M.; Paton, A.; Paton, J.; Jennings, M.
2014Preconditioning with endoplasmic reticulum stress ameliorates endothelial cell inflammationLeonard, A.; Paton, A.; El-Quadi, M.; Paton, J.; Fazal, F.; Komarova, Y.
2015IRE1α is an endogenous substrate of endoplasmic-reticulum-associated degradationSun, S.; Shi, G.; Sha, H.; Ji, Y.; Han, X.; Shu, X.; Ma, H.; Inoue, T.; Gao, B.; Kim, H.; Bu, P.; Guber, R.; Shen, X.; Lee, A.; Iwawaki, T.; Paton, A.; Paton, J.; Fang, D.; Tsai, B.; Yates, J.; et al.