Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/33909
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dc.contributor.authorRoediger, W.-
dc.contributor.authorGibbons, G.-
dc.date.issued2005-
dc.identifier.citationJournal of Neurology, 2005; 252(11):1418-1419-
dc.identifier.issn0340-5354-
dc.identifier.issn1432-1459-
dc.identifier.urihttp://hdl.handle.net/2440/33909-
dc.description© Steinkopff-
dc.description.statementofresponsibilityW.E.W. Roediger and G.F. Gibbons-
dc.language.isoen-
dc.publisherDr Dietrich Steinkopff Verlag-
dc.source.urihttp://dx.doi.org/10.1007/s00415-005-0869-x-
dc.subjectCytoplasm-
dc.subjectAnimals-
dc.subjectHumans-
dc.subjectDemyelinating Diseases-
dc.subjectNitric Oxide-
dc.subjectCoenzyme A-
dc.subjectModels, Neurological-
dc.subjectLipogenesis-
dc.titleInflammatory demyelination could be attributed to nitric oxide inhibition of cytosolic CoA with failed lipogenesis-
dc.typeJournal article-
dc.identifier.doi10.1007/s00415-005-0869-x-
pubs.publication-statusPublished-
dc.identifier.orcidRoediger, W. [0000-0002-6480-4804]-
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