Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/34728
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dc.contributor.authorPaton, J.-
dc.contributor.authorPaton, A.-
dc.date.issued2006-
dc.identifier.citationKidney International, 2006; 70(12):2049-2051-
dc.identifier.issn0085-2538-
dc.identifier.issn1523-1755-
dc.identifier.urihttp://hdl.handle.net/2440/34728-
dc.description© 2007 International Society of Nephrology-
dc.description.abstractThe pathway and the efficiency of intracellular trafficking of Shiga toxin differ between cell types, and this impacts on susceptibility to cytotoxicity. Warnier et al. demonstrate that in cell types targeted during human disease, Shiga toxin undergoes retrograde transport via the trans-Golgi network to the endoplasmic reticulum, albeit less efficiently than in HeLa cells.-
dc.description.statementofresponsibilityJ C Paton and A W Paton-
dc.language.isoen-
dc.publisherBlackwell Publishing Inc-
dc.source.urihttp://www.nature.com/ki/journal/v70/n12/abs/5001954a.html-
dc.subjectKidney-
dc.subjectCells, Cultured-
dc.subjectHumans-
dc.subjectShiga Toxins-
dc.subjectBiological Transport-
dc.titleShiga toxin 'goes retro' in human primary kidney cells-
dc.typeJournal article-
dc.identifier.doi10.1038/sj.ki.5001954-
pubs.publication-statusPublished-
dc.identifier.orcidPaton, J. [0000-0001-9807-5278]-
Appears in Collections:Aurora harvest 6
Molecular and Biomedical Science publications

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