Please use this identifier to cite or link to this item:
https://hdl.handle.net/2440/34728
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Full metadata record
DC Field | Value | Language |
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dc.contributor.author | Paton, J. | - |
dc.contributor.author | Paton, A. | - |
dc.date.issued | 2006 | - |
dc.identifier.citation | Kidney International, 2006; 70(12):2049-2051 | - |
dc.identifier.issn | 0085-2538 | - |
dc.identifier.issn | 1523-1755 | - |
dc.identifier.uri | http://hdl.handle.net/2440/34728 | - |
dc.description | © 2007 International Society of Nephrology | - |
dc.description.abstract | The pathway and the efficiency of intracellular trafficking of Shiga toxin differ between cell types, and this impacts on susceptibility to cytotoxicity. Warnier et al. demonstrate that in cell types targeted during human disease, Shiga toxin undergoes retrograde transport via the trans-Golgi network to the endoplasmic reticulum, albeit less efficiently than in HeLa cells. | - |
dc.description.statementofresponsibility | J C Paton and A W Paton | - |
dc.language.iso | en | - |
dc.publisher | Blackwell Publishing Inc | - |
dc.source.uri | http://www.nature.com/ki/journal/v70/n12/abs/5001954a.html | - |
dc.subject | Kidney | - |
dc.subject | Cells, Cultured | - |
dc.subject | Humans | - |
dc.subject | Shiga Toxins | - |
dc.subject | Biological Transport | - |
dc.title | Shiga toxin 'goes retro' in human primary kidney cells | - |
dc.type | Journal article | - |
dc.identifier.doi | 10.1038/sj.ki.5001954 | - |
pubs.publication-status | Published | - |
dc.identifier.orcid | Paton, J. [0000-0001-9807-5278] | - |
Appears in Collections: | Aurora harvest 6 Molecular and Biomedical Science publications |
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