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Results 1-10 of 13 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
2018Absence of vitamin D receptor in mature osteoclasts results in altered osteoclastic activity and bone lossStarczak, Y.; Reinke, D.; Barratt, K.; Ryan, J.; St-Arnaud, R.; Morris, H.; Davey, R.; Atkins, G.; Anderson, P.
2018Both ligand and VDR expression levels critically determine the effect of 1α,25-dihydroxyvitamin-D₃ on osteoblast differentiationYang, D.; Anderson, P.; Wijenayaka, A.; Barratt, K.; Triliana, R.; Stapledon, C.; Zhou, H.; Findlay, D.; Morris, H.; Atkins, G.
2016Evidence for altered osteoclastogenesis in splenocyte cultures from Cyp27b1 knockout miceReinke, D.; Kogawa, M.; Barratt, K.; Morris, H.; Anderson, P.; Atkins, G.
2016Early response of the human SOST gene to stimulation by 1α,25-dihydroxyvitamin D₃Wijenayaka, A.; Prideaux, M.; Yang, D.; Morris, H.; Findlay, D.; Anderson, P.; Atkins, G.
2014The local production of 1,25(OH)₂D₃ promotes osteoblast and osteocyte maturationTurner, A.; Hanrath, M.; Morris, H.; Atkins, G.; Anderson, P.; Bikle, D.; Norman, A.; Welsh, J.; 16th Vitamin D Workshop (11 Jun 2013 - 14 Jun 2013 : San Francisco, CA, USA)
2013Novel targets of vitamin D activity in bone: action of the vitamin D receptor in osteoblasts, osteocytes and osteoclastsRyan, J.; Reinke, D.; Kogawa, M.; Turner, A.; Atkins, G.; Anderson, P.; Morris, H.
2014Analysis of vitamin D metabolism gene expression in human bone: evidence for autocrine control of bone remodellingOrmsby, R.; Findlay, D.; Kogawa, M.; Anderson, P.; Morris, H.; Atkins, G.; Bikle, D.; Norman, A.; Welsh, J.; 16th Vitamin D Workshop (11 Jun 2013 - 14 Jun 2013 : San Francisco, CA, USA)
2011Vitamin D metabolism within bone cells: Effects on bone structure and strengthAnderson, P.; Atkins, G.; Turner, A.; Kogawa, M.; Findlay, D.; Morris, H.
2010The metabolism of 25-(OH)vitamin D3 by osteoclasts and their precursors regulates the differentiation of osteoclastsKogawa, M.; Anderson, P.; Findlay, D.; Morris, H.; Atkins, G.
2011The role of the calcitonin receptor in protecting against induced hypercalcemia is mediated via its actions in osteoclasts to inhibit bone resorptionTurner, A.; Tjahyono, F.; Chiu, W.; Skinner, J.; Sawyer, R.; Moore, A.; Morris, H.; Findlay, D.; Zajac, J.; Davey, R.