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Issue Date
Title
Author(s)
2012
Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors
Nieborowska-Skorska, M.
;
Kopinski, P.
;
Ray, R.
;
Hoser, G.
;
Ngaba, D.
;
Flis, S.
;
Cramer, K.
;
Reddy, M.
;
Koptyra, M.
;
Penserga, T.
;
Glodkowska-Mrowka, E.
;
Bolton, E.
;
Holyoake, T.
;
Eaves, C.
;
Cerny-Reiterer, S.
;
Valent, P.
;
Hochhaus, A.
;
Hughes, T.
;
Van der Kuip, H.
;
Sattler, M.
;
et al.
2009
Impact of baseline BCR-ABL mutations on response to Nilotinib in patients with chronic myeloid leukemia in chronic phase
Hughes, T.
;
Saglio, G.
;
Branford, S.
;
Soverini, S.
;
Kim, J.
;
Muller, M.
;
Martinelli, G.
;
Cortes, J.
;
Beppu, L.
;
Gottardi, E.
;
Dongho, K.
;
Erben, P.
;
Shou, Y.
;
Haque, A.
;
Gallagher, N.
;
Radich, J.
;
Hochhaus, A.
2010
Establishment of the first World Health Organization International Genetic Reference Panel for quantitation of BCR-ABL mRNA
Branford, S.
;
Hughes, T.
2009
Selecting optimal second-line tyrosine kinase inhibitor therapy for chronic myeloid leukemia patients after imatinib failure: does the BCR-ABL mutation status really matter?
Branford, S.
;
Vaz de Melo, J.
;
Hughes, T.
2009
Dasatinib treatment of chronic-phase chronic myeloid leukemia: analysis of responses according to preexisting BCR-ABL mutations
Muller, M.
;
Cortes, J.
;
Kim, D.
;
Druker, B.
;
Erben, P.
;
Pasquini, R.
;
Branford, S.
;
Hughes, T.
;
Radich, J.
;
Ploughman, L.
;
Mukhopadhyay, J.
;
Hochhaus, A.
2012
BCR-ABL1 doubling times more reliably assess the dynamics of CML relapse compared with the BCR-ABL1 fold rise: implications for monitoring and management
Branford, S.
;
Yeung, D.
;
Prime, J.
;
Choi, S.
;
Bang, J.
;
Park, J.
;
Kim, D.
;
Ross, D.
;
Hughes, T.
2005
In vitro sensitivity to imatinib-induced inhibition of ABL kinase activity is predictive of molecular response in patients with de-novo CML.
White, D.
;
Saunders, V.
;
Lyons, A.
;
Branford, S.
;
Grigg, A.
;
To, L.
;
Hughes, T.
2011
Sensitive detection of BCR-ABL1 mutations in patients with chronic myeloid leukemia after Imatinib resistance is predictive of outcome during subsequent therapy
Parker, W.
;
Lawrence, R.
;
Ho, M.
;
Irwin, D.
;
Scott, H.
;
Hughes, T.
;
Branford, S.
2012
Classification of patients with chronic myeloid leukemia on basis of BCR-ABL transcript level at 3 months fails to identify patients with low organic cation transporter-1 activity destined to have poor imatinib response
White, D.
;
Hughes, T.
2004
Real-time quantitative PCR analysis can be used as a primary screen to identify patients with CML treated with imatinib who have BCR-ABL kinase domain mutations
Branford, S.
;
Rudzki, Z.
;
Parkinson, I.
;
Grigg, A.
;
Taylor, K.
;
Seymour, J.
;
Durrant, S.
;
Browett, P.
;
Schwarer, A.
;
Arthur, C.
;
Catalano, J.
;
Leahy, M.
;
Filshie, R.
;
Bradstock, K.
;
Herrmann, R.
;
Joske, D.
;
Lynch, K.
;
Hughes, T.
Discover
Author
23
Branford, S.
14
Hochhaus, A.
8
White, D.
7
Kantarjian, H.
7
Kim, D.
7
Radich, J.
7
Saglio, G.
6
Cortes, J.
6
Saunders, V.
5
Grigg, A.
.
next >
Subject
34
Leukemia, Myelogenous, Chronic, B...
31
Pyrimidines
29
Imatinib Mesylate
29
Piperazines
28
Benzamides
23
Antineoplastic Agents
14
Protein Kinase Inhibitors
12
Drug Resistance, Neoplasm
12
Mutation
11
Male
.
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Date issued
19
2010 - 2017
24
2000 - 2009