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Results 21-30 of 36 (Search time: 0.003 seconds).
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PreviewIssue DateTitleAuthor(s)
2015Identification of markers that functionally define a quiescent multiple myeloma cell sub-population surviving bortezomib treatmentAdomako, A.; Calvo, V.; Biran, N.; Osman, K.; Chari, A.; Paton, J.; Paton, A.; Moore, K.; Schewe, D.; Aguirre-Ghiso, J.
2007Pathologic changes in mice induced by subtilase cytotoxin, a potent new Escherichia coli ABâ‚… toxin that targets the endoplasmic reticulumWang, H.; Paton, J.; Paton, A.
1995Increased oral virulence of Escherichia coli expressing a variant Shiga-like toxin type II operon is associated with both A subunit residues Met4 and Gly102Paton, A.; Manning, P.; Paton, J.
2001Characterization of Saa, a novel autoagglutinating adhesin produced by locus of enterocyte effacement-negative shiga-toxigenic Escherichia coli strains that are virulent for humansPaton, A.; Srimanote, P.; Woodrow, M.; Paton, J.
2000A new biological agent for treatment of Shiga toxigenic Escherichia coli infections and dysentery in humansPaton, A.; Morona, R.; Paton, J.
1995Comparative toxicity and virulence of Escherichia coli clones expressing variant and chimeric Shiga-like toxin type II operonsPaton, A.; Bourne, A.; Manning, P.; Paton, J.
2016The variable region of pneumococcal pathogenicity island 1 is responsible for unusually high virulence of a serotype 1 isolateHarvey, R.; Trappetti, C.; Mahdi, L.; Wang, H.; McAllister, L.; Scalvini, A.; Paton, A.; Paton, J.; Pirofski, L.
2017Autoinducer 2 signaling via the phosphotransferase FruA drives galactose utilization by streptococcus pneumoniae, resulting in hypervirulenceTrappetti, C.; McAllister, L.; Chen, A.; Wang, H.; Paton, A.; Oggioni, M.; McDevitt, C.; Paton, J.; McDaniel, L.
2014Opposing unfolded-protein-response signals converge on death receptor 5 to control apoptosisLu, M.; Lawrence, D.; Marsters, S.; Acosta-Alvear, D.; Kimmig, P.; Mendez, A.; Paton, A.; Paton, J.; Walter, P.; Ashkenazi, A.
2009Acquisition of Anergy to Proinflammatory Cytokines in Nonimmune Cells through Endoplasmic Reticulum Stress Response: A Mechanism for Subsidence of InflammationHayakawa, K.; Hiramatsu, N.; Okamura, M.; Yamazaki, H.; Nakajima, S.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.