Please use this identifier to cite or link to this item:
Scopus Web of ScienceĀ® Altmetric
Type: Journal article
Title: Mapping of a QTL contributing to cereal cyst nematode tolerance and resistance in wheat
Author: Williams, K.
Lewis, J.
Bogacki, P.
Pallotta, M.
Willsmore, K.
Kuchel, H.
Wallwork, H.
Citation: Australian Journal of Agricultural Research, 2003; 54(8):731-737
Publisher: C S I R O Publishing
Issue Date: 2003
ISSN: 0004-9409
Statement of
K. J. Williams, J. G. Lewis, P. Bogacki, M. A. Pallotta, K. L. Willsmore, H. Kuchel and H. Wallwork
Abstract: <jats:p>Cereal cyst nematode (CCN; Heterodera avenae Woll.) is a root pathogen of cereals that can cause severe yield losses in intolerant wheat cultivars. Tolerance to CCN, measured as early vigour in CCN-infested plots, was mapped in a Trident/Molineux doubled-haploid (DH) population. A locus accounting for a significant proportion of the tolerance to CCN was mapped to chromosome 6B of Molineux by association with RFLP loci Xcdo347-6B and Xbcd1 and also by nullisomic/tetrasomic substitution line analysis, and has been designated Cre8. The linkage of CCN tolerance with Xcdo347-6B was validated using a Barunga/Suneca DH population. The Cre8 locus also contributed to CCN resistance in the Trident/Molineux population. The RFLP locus Xbcd175, which is diagnostic for the Aegilops ventricosa segment VPM1 of Trident, explained up to 18% of the variation for early vigour in CCN-infested soils in the Trident/Molineux population. However, the Trident/Molineux population also segregated for early vigour in the absence of CCN, with Xbcd175 explaining up to 7% of the variation for this trait. The VPM1 segment of Trident therefore provides early vigour that may contribute to CCN tolerance in this cultivar.</jats:p>
RMID: 0020030480
DOI: 10.1071/AR02225
Appears in Collections:Agriculture, Food and Wine publications

Files in This Item:
There are no files associated with this item.

Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.