Please use this identifier to cite or link to this item: http://hdl.handle.net/2440/73782
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dc.contributor.authorRayner, C.en
dc.contributor.authorJones, K.en
dc.contributor.authorHorowitz, M.en
dc.date.issued2012en
dc.identifier.citationGastroparesis: Pathophysiology, Presentation and Treatment, 2012 / Parkman, H., McCallum, R. (ed./s), pp.177-190en
dc.identifier.isbn1607615517en
dc.identifier.isbn9781607615514en
dc.identifier.urihttp://hdl.handle.net/2440/73782-
dc.descriptionAlso has ISBN 1607615525 ; 9781607615521en
dc.description.abstractGastric emptying is frequently disordered in patients with long-standing type 1 and type 2 diabetes mellitus. While patients with diabetes present with similar symptoms to those with idiopathic gastroparesis, the relationship between gastric emptying and glycemic control represents an important additional consideration in the diabetic group. Specifically, acute variations in the blood glucose concentration affect the rate of gastric emptying, while conversely, gastric emptying is a key determinant of postprandial blood glucose. This has implications both for how gastroparesis should be defined in patients with diabetes, and for its optimal management.en
dc.description.statementofresponsibilityChristopher K. Rayner, Karen L. Jones and Michael Horowitzen
dc.language.isoenen
dc.publisherSpringeren
dc.subjectDiabetes mellitus; Interstitial cells of Cajal; Nitric oxide; Heme oxygenase 1; Incretin Glucagon-like peptide 1; insulin; blood glucose concentrationen
dc.titleDiabetic Gastroparesisen
dc.typeBook chapteren
dc.identifier.rmid0020119603en
dc.identifier.doi10.1007/978-1-60761-552-1_16en
dc.publisher.placeNew Yorken
dc.identifier.pubid24264-
pubs.library.collectionMedicine publicationsen
pubs.verification-statusVerifieden
pubs.publication-statusPublisheden
dc.identifier.orcidRayner, C. [0000-0002-5527-256X]en
dc.identifier.orcidJones, K. [0000-0002-1155-5816]en
dc.identifier.orcidHorowitz, M. [0000-0002-0942-0306]en
Appears in Collections:Medicine publications

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