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Type: Book chapter
Title: Dietary zinc supplementation and prenatal ethanol exposure
Author: Coyle, P.
Pearce, B.
Cowley, C.
Rofe, A.
Citation: Alcohol, Nutrition, and Health Consequences, 2013 / Watson, R., Preedy, V., Zibadi, S. (ed./s), pp.155-180
Publisher: Springer
Publisher Place: United States
Issue Date: 2013
ISBN: 9781627030465
Statement of
Peter Coyle, Brooke Summers-Pearce, Carina J. Cowley, and Allan M. Rofe
Abstract: Exposure to alcohol during pregnancy is associated with an increased risk of spontaneous abortion, growth retardation, congenital malformations and central nervous system dysfunction [1, 2]. These negative birth outcomes, which are collectively referred to as fetal alcohol spectrum disorder (FASD), range in severity from full fetal alcohol syndrome (FAS) through milder although clinically significant forms which can affect physical and behavioural outcomes (i.e. alcohol-related birth defects (ARBD) and alcohol-related neurodevelopmental disorders (ARND)). These outcomes are associated not only with chronic consumption of alcohol at high intakes and frequency but also with a single episode of alcohol intake, which is commonly called ‘binge drinking’ (>4 drinks/occasion). Although abstinence from alcohol during pregnancy would prevent these disorders, the motivation for self-restraint from drinking alcohol is not uniformly accepted among women [3–6]. Moreover, up to two-thirds of pregnancies are reported to be unplanned, indicating that many women may be unaware of their pregnancy when consuming alcohol [3, 5, 6]. Thus, the consumption of alcohol during pregnancy will continue to negatively impact on birth outcomes well into the future.
Keywords: Zinc; Pregnancy; Zinc deficiency; Birth defects; Fetus; Metallothionein; Ethanol/alcohol; Infection; Zinc supplementation
Rights: © Springer Science+Business Media New York 2013
RMID: 0020130003
DOI: 10.1007/978-1-62703-047-2_13
Appears in Collections:Molecular and Biomedical Science publications

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