Kowanko, I.Ferrante, A.Clemente, G.Kumaratilake, L.2006-07-262006-07-261996Infection and Immunity, 1996; 64(8):3435-34370019-95671098-5522http://hdl.handle.net/2440/14640The cytokine tumor necrosis factor (TNF) plays the important role of priming neutrophils for increased antimicrobial activity. We now demonstrate that human neutrophils which lack the ability to generate oxygen radicals, from patients with chronic granulomatous disease, show TNF-induced enhancement of killing of intraerythrocytic stages of Plasmodium falciparum but not of Staphylococcus aureus.enCopyright © 1996, American Society for MicrobiologyErythrocytesAnimalsHumansPlasmodium falciparumStaphylococcus aureusGranulomatous Disease, ChronicReactive Oxygen SpeciesTumor Necrosis Factor-alphaFree Radical ScavengersNeutrophil ActivationJournal article0030003361001996402110.1128/iai.64.8.3435-3437.1996A1996UY893000832-s2.0-003005589667367Ferrante, A. [0000-0002-2581-6407]