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Results 11-19 of 19 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
2012Caspase-2 deficiency promotes aberrant DNA-damage response and genetic instabilityDorstyn, L.; Puccini, J.; Wilson, C.; Shalini, S.; Nicola, M.; Moore, S.; Kumar, S.
2013Tropomyosin regulates cell migration during skin wound healingLees, J.; Ching, Y.; Adams, D.; Bach, C.; Samuel, M.; Kee, A.; Hardeman, E.; Gunning, P.; Cowin, A.; O'Neill, G.
2018Adult mouse eIF2Bϵ Arg191His astrocytes display a normal integrated stress response in vitroWisse, L.; Ter Braak, T.; Van De Beek, M.; Van Berkel, C.; Wortel, J.; Heine, V.; Proud, C.; Van Der Knaap, M.; Abbink, T.
2011TRPA1 contributes to specific mechanically activated currents and sensory neuron mechanical hypersensitivityBrierley, S.; Castro Kraftchenko, J.; Harrington, A.; Hughes, P.; Page, A.; Rychkov, G.; Blackshaw, L.
2012Sphingosine-1-phosphate facilitates trafficking of hematopoietic stem cells and their mobilization by CXCR4 antagonists in miceJuarez, J.; Harun, N.; Thien, M.; Welschinger, R.; Baraz, R.; Dela Pena, A.; Pitson, S.; Rettig, M.; DiPersio, J.; Bradstock, K.; Bendall, L.
2010Genetic dissection of differential signaling threshold requirements for the Wnt/β-Catenin pathway in vivoBuchert, M.; Athineos, D.; Abud, H.; Burke, Z.; Faux, M.; Samuel, M.; Jarnicki, A.; Winbanks, C.; Newton, I.; Meniel, V.; Suzuki, H.; Stacker, S.; Nathke, I.; Tosh, D.; Huelsken, J.; Clarke, A.; Heath, J.; Sansom, O.; Ernst, M.; Nusse, R.
2011DNMT3L is a regulator of X chromosome compaction and post-meiotic gene transcriptionZamudio, N.; Scott, H.; Wolski, K.; Lo, C.; Law, C.; Leong, D.; Kinkel, S.; Chong, S.; Jolley, D.; Smyth, G.; De Kretser, D.; Whitelaw, E.; O'Bryan, M.; Pera, M.
2016Pcdh19 loss-of-function increases neuronal migration in vitro but is dispensable for brain development in micePederick, D.; Homan, C.; Jaehne, E.; Piltz, S.; Haines, B.; Baune, B.; Jolly, L.; Hughes, J.; Gecz, J.; Thomas, P.
2013MHC class II-alpha chain knockout mice support increased viral replication that is independent of their lack of MHC class II cell surface expression and associated immune function deficienciesAlsharifi, M.; Koskinen, A.; Wijesundara, D.; Bettadapura, J.; Mullbacher, A.; Ashour, H.M.