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https://hdl.handle.net/2440/80752
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dc.contributor.author | O'Donnell, M. | - |
dc.contributor.author | Varker, T. | - |
dc.contributor.author | Creamer, M. | - |
dc.contributor.author | Fletcher, S. | - |
dc.contributor.author | McFarlane, A. | - |
dc.contributor.author | Silove, D. | - |
dc.contributor.author | Bryant, R. | - |
dc.contributor.author | Forbes, D. | - |
dc.date.issued | 2013 | - |
dc.identifier.citation | Psychosomatic Medicine, 2013; 75(1):68-75 | - |
dc.identifier.issn | 0033-3174 | - |
dc.identifier.issn | 1534-7796 | - |
dc.identifier.uri | http://hdl.handle.net/2440/80752 | - |
dc.description.abstract | OBJECTIVE The first aim of this work was to conduct a rigorous longitudinal study to identify rates of delayed-onset posttraumatic stress disorder (PTSD) in a sample of patients with severe injury. The second aim was to determine what variables differentiated delayed-onset PTSD from chronic PTSD. METHODS Randomly selected patients with injury who were admitted to four hospitals around Australia were recruited to the study (N = 834) and assessed in the acute care hospital, at 3 months, and at 12 months. A structured clinical interview was used to assess PTSD at each time point. RESULTS Seventy-three patients (9%; n = 73) had PTSD at 12 months. Of these, 39 (53%) were classified as having delayed-onset PTSD. Furthermore, 22 (56%) patients with delayed-onset PTSD had minimal PTSD symptoms at 3 months (i.e., they did not have partial/subsyndromal PTSD at 3 months). The variables that differentiated delayed-onset PTSD from chronic PTSD were greater injury severity (odds ratio [OR] = 1.13; 95% confidence interval [CI] = 1.02–1.26), lower anxiety severity at 3 months (OR = 0.73; 95% CI = 0.61–0.87), and greater pain severity at 3 months (OR = 1.39; 95% CI = 1.06–1.84). CONCLUSIONS Delayed-onset PTSD occurred frequently in this sample. Approximately half of the patients with delayed-onset PTSD had minimal PTSD symptoms at 3 months; therefore, their delayed-onset PTSD could not be accounted for by a small number of fluctuating symptoms. As we move toward DSM-V, it is important that research continues to explore the factors that underpin the development of delayed-onset PTSD. | - |
dc.description.statementofresponsibility | Meaghan L. O’Donnell, Tracey Varker, Mark Creamer, Susan Fletcher, Alexander C. McFarlane, Derrick Silove, Richard A. Bryant, David Forbes | - |
dc.language.iso | en | - |
dc.publisher | Lippincott Williams & Wilkins | - |
dc.rights | Copyright © 2013 by the American Psychosomatic Society | - |
dc.source.uri | http://www.psychosomaticmedicine.org/content/75/1/68 | - |
dc.subject | delayed onset | - |
dc.subject | posttraumatic stress disorder | - |
dc.subject | subsyndromal PTSD | - |
dc.subject | partial PTSD | - |
dc.subject | prediction | - |
dc.title | Exploration of delayed-onset posttraumatic stress disorder after severe injury | - |
dc.type | Journal article | - |
dc.identifier.doi | 10.1097/PSY.0b013e3182761e8b | - |
dc.relation.grant | http://purl.org/au-research/grants/nhmrc/568970 | - |
pubs.publication-status | Published | - |
dc.identifier.orcid | McFarlane, A. [0000-0002-3829-9509] | - |
Appears in Collections: | Aurora harvest 4 Medicine publications |
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