Circulating epigenomic biomarkers correspond with kidney disease susceptibility in high-risk populations with type 2 diabetes mellitus

Files

hdl_140221.pdf (3.86 MB)
  (Published version)

Date

2023

Authors

Khurana, I.
Howard, N.J.
Maxwell, S.
Du Preez, A.
Kaipananickal, H.
Breen, J.
Buckberry, S.
Okabe, J.
Al-Hasani, K.
Nakasatien, S.

Editors

Advisors

Journal Title

Journal ISSN

Volume Title

Type:

Journal article

Citation

Diabetes Research and Clinical Practice, 2023; 204:110918-1-110918-9

Statement of Responsibility

Ishant Khurana, Natasha J. Howard, Scott Maxwell, Anelle Du Preez, Harikrishnan Kaipananickal, James Breen, Sam Buckberry, Jun Okabe, Keith Al-Hasani, Soontaree Nakasatien, Thep Himathongkam, Mark E. Cooper, Louise Maple-Brown, Yotsapon Thewjitcharoen, Alex Brown, Assam El-Osta

Conference Name

Abstract

AIMS: To investigate epigenomic indices of diabetic kidney disease (DKD) susceptibility among high-risk populations with type 2 diabetes mellitus. METHODS: KDIGO (Kidney Disease: Improving Global Outcomes) clinical guidelines were used to classify people living with or without DKD. Differential gene methylation of DKD was then assessed in a discovery Aboriginal Diabetes Study cohort (PROPHECY, 89 people) and an external independent study from Thailand (THEPTARIN, 128 people). Corresponding mRNA levels were also measured and linked to levels of albuminuria and eGFR. RESULTS: Increased DKD risk was associated with reduced methylation and elevated gene expression in the PROPHECY discovery cohort of Aboriginal Australians and these findings were externally validated in the THEPTARIN diabetes registry of Thai people living with type 2 diabetes mellitus. CONCLUSIONS: Novel epigenomic scores can improve diagnostic performance over clinical modelling using albuminuria and GFR alone and can distinguish DKD susceptibility.

School/Discipline

Dissertation Note

Provenance

Description

Available online 24 September 2023

Access Status

Rights

© 2023 The Author(s). Published by Elsevier B.V. This is an open access article under the CC BY-NC license (http://creativecommons.org/licenses/bync/4.0/).

License

Call number

Persistent link to this record