Cross-resistance to imazethapyr in a fluazifop-P-butyl-resistant population of Digitaria sanguinalis

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dc.contributor.authorHidayat, I.
dc.contributor.authorPreston, C.
dc.date.issued2001
dc.descriptionCopyright © 2001 Elsevier Science (USA). All rights reserved.
dc.description.abstractA population of Digitaria sanguinalis selected by and resistant to the acetyl-coenzyme A carboxylase-inhibiting herbicide fluazifop-P-butyl is cross-resistant to the acetolactate synthase (ALS) inhibitor imazethapyr. The resistant population was six-fold resistant to imazethapyr despite never having been exposed to any ALS-inhibiting herbicide. ALS activity extracted from the resistant population was susceptible to imidazolinone herbicides. Malathion, a known cytochrome P450 monooxygenase inhibitor, synergized imazethapyr activity on the resistant population, suggesting that resistance is due to increased herbicide metabolism. In contrast, malathion antagonized fluazifop-P-butyl activity. Therefore, it is likely that the enzyme responsible for fluazifop acid detoxification in D. sanguinalis is different from the enzyme that detoxifies imazethapyr.
dc.description.statementofresponsibilityImam Hidayat and Christopher Preston
dc.description.urihttp://www.elsevier.com/wps/find/journaldescription.cws_home/622930/description#description
dc.identifier.citationPesticide Biochemistry and Physiology, 2001; 71(3):190-195
dc.identifier.doi10.1006/pest.2001.2576
dc.identifier.issn0048-3575
dc.identifier.orcidPreston, C. [0000-0002-7588-124X]
dc.identifier.urihttp://hdl.handle.net/2440/27773
dc.language.isoen
dc.publisherAcademic Press Inc
dc.source.urihttps://doi.org/10.1006/pest.2001.2576
dc.titleCross-resistance to imazethapyr in a fluazifop-P-butyl-resistant population of Digitaria sanguinalis
dc.typeJournal article
pubs.publication-statusPublished

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