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Results 31-40 of 41 (Search time: 0.003 seconds).
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PreviewIssue DateTitleAuthor(s)
2008Pneumococcal Virulence Gene Expression and Host Cytokine Profiles during Pathogenesis of Invasive DiseaseMahdi, L.; Ogunniyi, A.; Le Messurier, K.; Paton, J.
2007Contributions of pneumolysin, pneumococcal surface protein A (PspA), and PspC to pathogenicity of Streptococcus pneumoniae D39 in a mouse modelOgunniyi, A.; Le Messurier, K.; Graham, R.; Watt, J.; Briles, D.; Stroeher, U.; Paton, J.
2001Protection against Streptococcus pneumoniae elicited by immunization with pneumolysin and CbpAOgunniyi, A.; Woodrow, M.; Poolman, J.; Paton, J.; Tuomanen, E.I.
2013Site of isolation determines biofilm formation and virulence phenotypes of streptococcus pneumoniae serotype 3 clinical isolatesTrappetti, C.; van der Maten, E.; Amin, Z.; Potter, A.; Chen, A.; van Mourik, P.; Lawrence, A.; Paton, A.; Paton, J.; Camilli, A.
2002Role of RegM, a homologue of the catabolite repressor protein CcpA, in the virulence of Streptococcus pneumoniaeGiammarinaro, P.; Paton, J.
2000Immunization of mice with combinations of pneumococcal virulence proteins elicits enhanced protection against challenge with Streptococcus pneumoniaeOgunniyi, A.; Folland, R.; Briles, D.; Hollingshead, S.; Paton, J.; Tuomanen, E.I.
2011A variable region within the genome of Streptococcus pneumoniae contributes to strain-strain variation in virulenceHarvey, R.; Stroeher, U.; Ogunniyi, A.; Smith-Vaughan, H.; Leach, A.; Paton, J.; Neyrolles, O.
2011A molecular mechanism for bacterial susceptibility to zincMcDevitt, C.; Ogunniyi, A.; Valkov, E.; Lawrence, M.; Kobe, B.; McEwan, A.; Paton, J.; Imlay, J.
2011Extracellular matrix formation enhances the ability of streptococcus pneumoniae to cause invasive diseaseTrappetti, C.; Ogunniyi, A.; Oggioni, M.; Paton, J.; Miyaji, E.N.
2014Development of primary invasive pneumococcal disease caused by serotype 1 pneumococci is driven by early increased type I interferon response in the lungHughes, C.; Harvey, R.; Plumptre, C.; Paton, J.; Pirofski, L.